Joshua D. Schiffman

Primary Children's Hospital

Full Professor(heuristic)

Cancer-related Molecular Pathways · Oncology

h-index53

i10-index151

Publications478

Citations14,606

Grants

No NSF/NIH grants on record.

Career Trajectory

Research Cluster Map

Direct collaborators. Node size = influence, colour = community, dashed = past collaboration.

Potential Collaborators

Recent Publications

Last 2 years · showing 20 of 58

Figure S2 from Germline Pathogenic <i>DROSHA</i> Variants Are Linked to Pineoblastoma and Wilms Tumor Predisposition
2026-06-17
Figure S4 from Germline Pathogenic <i>DROSHA</i> Variants Are Linked to Pineoblastoma and Wilms Tumor Predisposition
2026-06-17
Figure S1 from Germline Pathogenic <i>DROSHA</i> Variants Are Linked to Pineoblastoma and Wilms Tumor Predisposition
2026-06-17
Figure S3 from Germline Pathogenic <i>DROSHA</i> Variants Are Linked to Pineoblastoma and Wilms Tumor Predisposition
2026-06-17
Figure S5 from Germline Pathogenic <i>DROSHA</i> Variants Are Linked to Pineoblastoma and Wilms Tumor Predisposition
2026-06-17
Abstract 7639: Primate comparative oncology reveal humans' unique cancer susceptibility
Cancer Research · 2026-04-03
Abstract 1157: Preclinical evaluation of PEG-[SN22]4 (PEEL-224), a multivalent polymeric camptothecin prodrug, in pediatric solid tumor patient-derived xenograft models
Cancer Research · 2026-04-03
Genome-wide association study meta-analysis identifies susceptibility loci informing Ewing sarcoma etiology and potential mechanisms of risk
medRxiv · 2026-02-09
Divergent understandings in comparative oncology
Proceedings of the National Academy of Sciences · 2026-02-02
Development and pilot testing of AYA-RISE, a risk information and screening education intervention for adolescents and young adults with cancer risk syndromes
Familial Cancer · 2026-01-23
Supplementary Figures from Li–Fraumeni Syndrome–Associated Dimer-Forming Mutant p53 Promotes Transactivation-Independent Mitochondrial Cell Death
2025-12-11
Supplementary Data 3 from Li–Fraumeni Syndrome–Associated Dimer-Forming Mutant p53 Promotes Transactivation-Independent Mitochondrial Cell Death
2025-12-11
Immune-cancer analyses across mammals reveal a potential trophic level and platelet-linked tradeoff between cancer and trauma mortalityPreprint
bioRxiv (Cold Spring Harbor Laboratory) · 2025-12-11 · 1 citations
Supplementary Table 1 from Li–Fraumeni Syndrome–Associated Dimer-Forming Mutant p53 Promotes Transactivation-Independent Mitochondrial Cell Death
2025-12-11
Supplementary Table 2 from Li–Fraumeni Syndrome–Associated Dimer-Forming Mutant p53 Promotes Transactivation-Independent Mitochondrial Cell Death
2025-12-11
Supplementary Data 1 from Li–Fraumeni Syndrome–Associated Dimer-Forming Mutant p53 Promotes Transactivation-Independent Mitochondrial Cell Death
2025-12-11
Supplementary Data 6 from Li–Fraumeni Syndrome–Associated Dimer-Forming Mutant p53 Promotes Transactivation-Independent Mitochondrial Cell Death
2025-12-11
Supplementary Data 5 from Li–Fraumeni Syndrome–Associated Dimer-Forming Mutant p53 Promotes Transactivation-Independent Mitochondrial Cell Death
2025-12-11
Supplementary Data 4 from Li–Fraumeni Syndrome–Associated Dimer-Forming Mutant p53 Promotes Transactivation-Independent Mitochondrial Cell Death
2025-12-11
Supplementary Data 2 from Li–Fraumeni Syndrome–Associated Dimer-Forming Mutant p53 Promotes Transactivation-Independent Mitochondrial Cell Death
2025-12-11

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