M Beer

Johns Hopkins University

Full Professor(heuristic)

Magnetic confinement fusion research · Nuclear and High Energy Physics

h-index56

i10-index126

Publications5,020

Citations18,947

Grants

No NSF/NIH grants on record.

Career Trajectory

Research Cluster Map

Direct collaborators. Node size = influence, colour = community, dashed = past collaboration.

Potential Collaborators

Recent Publications

Last 2 years · showing 20 of 49

CRISPRa Screening for PDX1 CCR in Human Embryonic Stem Cells v1
2026-05-31
CRISPRa screen analysis to identify CCRs associated with DE genes v1
2026-05-31
CRISPRa Perturb-Seq Screen v1
2026-05-31
CRISPRi Screening for PDX1 Enhancers in Pancreatic Progenitor Cells v1
2026-05-30
Steroid hormone antagonism affords vascular protection in a mouse model of vascular Ehlers-Danlos syndrome
JCI Insight · 2026-04-28
Reprogramming of neuronal genome function and phenotype by astrocytes
bioRxiv (Cold Spring Harbor Laboratory) · 2026-03-07
Figure S4 from The Histone Methyltransferase KMT2D Is a Critical Mediator of Lineage Plasticity and Therapeutic Response in Castration-Resistant Prostate Cancer
2026-03-02
Data from The Histone Methyltransferase KMT2D Is a Critical Mediator of Lineage Plasticity and Therapeutic Response in Castration-Resistant Prostate Cancer
2026-03-02
Figure S9 from The Histone Methyltransferase KMT2D Is a Critical Mediator of Lineage Plasticity and Therapeutic Response in Castration-Resistant Prostate Cancer
2026-03-02
Supplementary Data 1 from The Histone Methyltransferase KMT2D Is a Critical Mediator of Lineage Plasticity and Therapeutic Response in Castration-Resistant Prostate Cancer
2026-03-02
Figure S3 from The Histone Methyltransferase KMT2D Is a Critical Mediator of Lineage Plasticity and Therapeutic Response in Castration-Resistant Prostate Cancer
2026-03-02
Figure S7 from The Histone Methyltransferase KMT2D Is a Critical Mediator of Lineage Plasticity and Therapeutic Response in Castration-Resistant Prostate Cancer
2026-03-02
Supplementary Data 2 from The Histone Methyltransferase KMT2D Is a Critical Mediator of Lineage Plasticity and Therapeutic Response in Castration-Resistant Prostate Cancer
2026-03-02
Figure S6 from The Histone Methyltransferase KMT2D Is a Critical Mediator of Lineage Plasticity and Therapeutic Response in Castration-Resistant Prostate Cancer
2026-03-02
Figure S2 from The Histone Methyltransferase KMT2D Is a Critical Mediator of Lineage Plasticity and Therapeutic Response in Castration-Resistant Prostate Cancer
2026-03-02
Figure S10 from The Histone Methyltransferase KMT2D Is a Critical Mediator of Lineage Plasticity and Therapeutic Response in Castration-Resistant Prostate Cancer
2026-03-02
Figure S5 from The Histone Methyltransferase KMT2D Is a Critical Mediator of Lineage Plasticity and Therapeutic Response in Castration-Resistant Prostate Cancer
2026-03-02
Figure S1 from The Histone Methyltransferase KMT2D Is a Critical Mediator of Lineage Plasticity and Therapeutic Response in Castration-Resistant Prostate Cancer
2026-03-02
Figure S8 from The Histone Methyltransferase KMT2D Is a Critical Mediator of Lineage Plasticity and Therapeutic Response in Castration-Resistant Prostate Cancer
2026-03-02
Experimental procedures for TF Perturb-Seq (UT Southwestern, MSKCC) v1
2026-02-10

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