Steven E. Schumacher

Broad Institute

Full Professor(heuristic)

Cancer Genomics and Diagnostics · Cancer Research

h-index108

i10-index148

Publications342

Citations111,453

Grants

No NSF/NIH grants on record.

Career Trajectory

Research Cluster Map

Direct collaborators. Node size = influence, colour = community, dashed = past collaboration.

Potential Collaborators

Recent Publications

Last latest available · showing 20 of 134

Crowd-sourced benchmarking of single-sample tumor subclonal reconstruction
Nature Biotechnology · 2024-06-11 · 13 citations
Supplementary Table S2 from Systematic Interrogation of 3q26 Identifies <i>TLOC1</i> and <i>SKIL</i> as Cancer Drivers
2023-04-03
Data from <i>MECP2</i> Is a Frequently Amplified Oncogene with a Novel Epigenetic Mechanism That Mimics the Role of Activated RAS in MalignancyPreprint
2023-04-03
Supplementary Figures 1-6, Tables 1-2, Methods from Genetic and Functional Studies Implicate <i>HIF1</i>α as a 14q Kidney Cancer Suppressor GenePreprint
2023-04-03
Supplementary Table S5 from Systematic Interrogation of 3q26 Identifies <i>TLOC1</i> and <i>SKIL</i> as Cancer Drivers
2023-04-03
Supplementary Table S7 from Systematic Interrogation of 3q26 Identifies <i>TLOC1</i> and <i>SKIL</i> as Cancer Drivers
2023-04-03
Supplementary Data from <i>MECP2</i> Is a Frequently Amplified Oncogene with a Novel Epigenetic Mechanism That Mimics the Role of Activated RAS in MalignancyPreprint
2023-04-03
Supplementary Figures S1 - S6 from <i>MECP2</i> Is a Frequently Amplified Oncogene with a Novel Epigenetic Mechanism That Mimics the Role of Activated RAS in MalignancyPreprint
2023-04-03
Supplementary Tables S1 - S3 from <i>MECP2</i> Is a Frequently Amplified Oncogene with a Novel Epigenetic Mechanism That Mimics the Role of Activated RAS in Malignancy
2023-04-03
Supplementary Figures S1 - S6 from <i>MECP2</i> Is a Frequently Amplified Oncogene with a Novel Epigenetic Mechanism That Mimics the Role of Activated RAS in MalignancyPreprint
2023-04-03
Supplementary Figure 3 from Systematic Interrogation of 3q26 Identifies <i>TLOC1</i> and <i>SKIL</i> as Cancer DriversPreprint
2023-04-03
Supplementary Figure 2 from Systematic Interrogation of 3q26 Identifies <i>TLOC1</i> and <i>SKIL</i> as Cancer DriversPreprint
2023-04-03
Supplementary Table S3 from Systematic Interrogation of 3q26 Identifies <i>TLOC1</i> and <i>SKIL</i> as Cancer Drivers
2023-04-03
Supplementary Table S8 from Systematic Interrogation of 3q26 Identifies <i>TLOC1</i> and <i>SKIL</i> as Cancer Drivers
2023-04-03
Supplementary Tables S1 - S3 from <i>MECP2</i> Is a Frequently Amplified Oncogene with a Novel Epigenetic Mechanism That Mimics the Role of Activated RAS in Malignancy
2023-04-03
Supplementary Data from <i>MECP2</i> Is a Frequently Amplified Oncogene with a Novel Epigenetic Mechanism That Mimics the Role of Activated RAS in MalignancyPreprint
2023-04-03
Supplementary Methods from <i>MECP2</i> Is a Frequently Amplified Oncogene with a Novel Epigenetic Mechanism That Mimics the Role of Activated RAS in MalignancyPreprint
2023-04-03
Data from <i>MECP2</i> Is a Frequently Amplified Oncogene with a Novel Epigenetic Mechanism That Mimics the Role of Activated RAS in MalignancyPreprint
2023-04-03
Supplementary Methods from <i>MECP2</i> Is a Frequently Amplified Oncogene with a Novel Epigenetic Mechanism That Mimics the Role of Activated RAS in MalignancyPreprint
2023-04-03
Supplementary Legends from Systematic Interrogation of 3q26 Identifies <i>TLOC1</i> and <i>SKIL</i> as Cancer DriversPreprint
2023-04-03

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